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By Anca Maria Moldoveanu (editor)

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ISSN: 0140-6736. Brunekreef, B. (2007). Health effects of air pollution observed in cohort studies in Europe. Journal of Exposure Science and Environmental Epidemiology. S61-5. ISSN:1559-0631. Choi, H; Jedrychowski, W; Spengler, J; Camann, DE; Whyatt, RM; Rauh, V; Tsai, WY & Perera, FP. (2006). International studies of prenatal exposure to polycyclic aromatic hydrocarbons and fetal growth. Environmental Health Perspect. 1744-50. ISSN: 0091/6765. Darrow, LA; Klein, M; Flanders, WD; Waller, LA; Correa, A; Marcus, M; Mulholland, JA; Russell, AG & Tolbert, PE.

2006) performed a robust study on plausible fisiopathological mechanisms by which exposure to particulate matter may lead to adverse perinatal outcomes. They identified five mechanistic pathways including a) oxidative stress; b) pulmonary and placental inflammation; c) blood coagulation; d) alteration of endothelial function; and e) hemodynamic responses to particulate exposure. As Ritz & Wilhelm given attention (2008), these pathways may not act independently. For example, an increase in maternal blood pressure and an impaired trophoblast invasion of the spiral arteries may induce uteroplacental hypoperfusion and a state of relative hypoxia surrounding the trophoblast.

Air pollution and premature births 3. , 2011). Because of different exposure assessments, methods of ascertainment, measurement times and collinearity between pollutants, the results about the association showed heterogeneity and/or an absence of association. We summarized our systemic review of these research papers in tables 1 and 2.

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